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A) 1.0 B) 2.0 C) 2.5 D) 1.5
A) Ten times B) Three times C) Two times D) Five times
A) Two times B) Ten times C) Eight times D) Five times
A) Risk of internal hemorrhage B) Risk of vitamin K deficiency C) High risk of stent thrombosis D) Risk of INR fluctuation
A) Fresh frozen plasma B) Recombinant factor VIIa C) Prothrombin complex concentrates D) Vitamin K
A) High risk of infection B) Slow onset of action C) High cost of treatment D) High risk of thromboembolism
A) To reduce the time for rewarfarinisation B) To avoid liver damage C) To prevent allergic reactions D) To improve INR stability
A) Vitamin K B) Fresh frozen plasma C) Prothrombin complex concentrates D) Activated charcoal
A) 5% to 10% B) 20% to 30% C) 1% to 3% D) 10% to 20%
A) 327,000 B) 301,000 C) 400,000 D) 200,000
A) 40% B) 30% C) 20% D) 80%
A) 40% B) 70% C) 20% D) 50%
A) 80% B) 40% C) 70% D) 20%
A) Airborne droplets B) Sharing food utensils C) Direct skin contact D) Mosquito bites
A) Elimination of sugar from diets B) Increased smoking cessation C) New effective acid suppressants D) Widespread vaccination
A) Rational use of NSAIDs B) Increased intake of vitamin K C) Increased use of anticoagulants D) Higher reliance on warfarin
A) Cheaper in cost B) More effective at preventing thromboembolism C) Easier to reverse with vitamin K D) Fewer interactions with food
A) Esophageal ulcer B) Meckel's diverticulum ulcer C) Gastric ulcer D) Duodenal ulcer
A) Type II B) Type III C) Type V D) Type I
A) Perpendicular with no surrounding inflammation B) Smooth with no elevation C) Irregular with elevated borders D) Regular with elevated borders
A) Epithelial tissue, muscle tissue, blood vessels, and nerves B) Fibrin, collagen, epithelial cells, and inflammatory exudate C) Granuloma, fibrous tissue, necrotic debris, and mucus D) Fibrinoid necrosis, inflammatory exudate, granulation tissue, and fibrous tissue
A) Proton pump inhibitor (PPI) B) Aspirin C) H2 antagonist D) Misoprostol
A) It promotes abortion and causes gastrointestinal distress B) It has high cardiovascular risks C) It interacts negatively with proton pump inhibitors D) It is ineffective against gastric ulcers
A) Increased dosage of amoxicillin B) Immediate surgery C) Bismuth-containing quadruple therapy D) Triple regimen therapy
A) 60% B) 70% C) 90% D) 80%
A) Type I B) Type II C) Type III D) Type IV
A) Muscularis mucosae and lamina propria B) Serosa and muscularis propria C) Submucosa and adventitia D) Epithelium and basement membrane
A) Naproxen with PPI B) Ibuprofen with PPI C) Aspirin with H2 antagonist D) Celecoxib with misoprostol
A) It increases B) It drops below 50% C) It decreases D) It stays the same
A) Elevated and regular B) Perpendicular C) Irregular D) Smooth
A) Increased appetite B) Catastrophic consequences C) Immediate healing D) Chronic constipation
A) Acute chemical peritonitis B) Pulmonary embolism C) Systemic hypoglycemia D) Viral gastroenteritis
A) Sudden intense abdominal pain B) Severe diarrhea C) High fever D) Blurred vision
A) Valentino's syndrome B) Cushing's syndrome C) Conn's syndrome D) Zollinger-Ellison syndrome
A) 10% B) 5% C) 20% D) 50%
A) A narrowing of the pyloric canal B) The spread of bacteria to the lungs C) The thickening of the stomach wall D) A hole leading into adjacent organs
A) Stenosis B) Hyperchlorhydria C) Metaplasia D) Peritonitis
A) Severe vomiting B) High fever C) Internal bleeding D) Severe constipation
A) Stool culture B) Biopsies C) Blood tests D) Physical examination
A) Lipase B) Amylase C) Pepsin D) Urease
A) Glucagon B) Cortisol C) BabA D) Insulin
A) VacA B) CagA C) OipA D) PicB
A) Lack of stomach acid B) Viral infection C) An acidic environment D) An alkaline environment
A) BabA B) IL1B C) TNF D) CagA
A) The gallbladder B) The stomach C) The esophagus D) The small intestine
A) Lack of physical exercise B) Chronic dehydration C) Excessive intake of spicy foods D) Helicobacter pylori infection
A) Urine analysis B) Liver function test C) MRI scan D) Urea breath test
A) For all gastric ulcer cases B) When endoscopic treatment is unsuccessful C) Only in elderly patients D) As the first line of treatment
A) They neutralize stomach acid B) They can cause or worsen ulcers C) They help heal the stomach lining D) They kill H. pylori bacteria
A) Teenagers B) Young adults C) Older people D) Children
A) Nausea B) Waterbrash C) Heartburn D) Belching
A) COX-1 B) COX-2 C) Lipoxygenase D) Pepsinogen
A) Physiological stress from serious health problems B) Excessive spice consumption C) High caffeine intake D) Chronic psychological stress
A) Blood antibody test B) Esophagogastroduodenoscopy (EGD) C) Barium contrast x-ray D) Stool antigen test
A) Enlargement of the stomach lining B) Presence of H. pylori colonies C) Free gas underneath the diaphragm D) Increased blood flow to the stomach
A) Anticoagulants B) Antibiotics C) Proton-pump inhibitors D) Antacids
A) Nitrogen gas B) Radioactive carbon C) Barium sulfate D) Hydrochloric acid
A) Over age 45 B) Over age 30 C) Over age 60 D) Over age 18
A) Carcinoid B) Lymphoma C) Adenocarcinoma D) Gastrinoma
A) Severe allergic reaction B) A false negative result C) A false positive result D) A highly accurate result
A) Less than 5 g/dL B) Greater than 12 g/dL C) Greater than 7 g/dL D) Exactly 10 g/dL
A) Glasgow-Blatchford score B) MELD score C) Child-Pugh score D) APACHE II score
A) Antifibrinolytics B) Crystalloids C) Antacids D) Prokinetic agents
A) 20 mg bolus followed by 2 mg per hour B) 80 mg bolus followed by 8 mg per hour C) 40 mg bolus followed by 4 mg per hour D) 100 mg bolus followed by 10 mg per hour
A) 96 hours B) 48 hours C) 24 hours D) 72 hours
A) Antifibrinolytic agents B) Tranexamic acid C) Cautery D) Oral PPIs
A) 1958 B) 1997 C) 1984 D) 1982
A) Nausea B) Halitosis C) Vomiting D) Severe abdominal pain
A) Journal of Molecular Biology B) Karolinska Medical Review C) Australian Medical Journal D) New England Medical Journal
A) 1997 B) 1984 C) 2005 D) 1998
A) Athens, Greece B) UWA in Perth, Western Australia C) Atlanta, Georgia D) Stockholm, Sweden
A) Mastic gum B) A petri dish culture C) Spicy food extracts D) Antibiotics
A) Moderate improvement B) No effect C) Immediate elimination D) High effectiveness
A) Microbiologist B) General practitioner C) Nobel Laureate D) Gastroenterologist |